摘要:类风湿关节炎(rheumatoid arthritis,RA)是一种高度致残性自身免疫性疾病,其基本特征是炎症性滑膜炎伴关节软骨和骨的破坏,破骨细胞在RA骨质破坏的病理机制中发挥着重要作用。RANKL/RANK/OPG系统是近年来发现的与破骨细胞分化及活化过程密切相关的细胞因子,包括:核因子κB受体激活配体(RANKL)(receptor activator of NF—κB ligand),
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