摘要:大量研究发现:糖尿病的机体大多处于炎症或者氧化应激状态,并多伴有炎症因子及其他应激分子水平的升高,从而激活C—jun氨基端激酶(JNK)信号转导通路,参与糖尿病慢性并发症的发生和(或)发展。同时更多进一步的研究还表明选择性抑制JNK的激活将可能为糖尿病并发症治疗提供一个新方向。以下就该领域目前的研究做一综述。
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